Cover Image

HARDBACK
$54.00



View/Hide Left Panel

Sources of environmental arsenic include smelters, electric power plants using arsenic-rich coal, and soil and water found in certain parts of the world.18

Human poisoning from the burning of CCA-impregnated wood has not been previously reported and represents the probable source of arsenic exposure in this family. Fowler18 warned in 1977 that the burning of CCA-treated wood should be studied as a potential health hazard. Our studies of the kitchen-living area were done in the summer and disclosed notable contamination with CCA-rich ash. In the winter months while the CCA-treated plywood was being burned, we would have anticipated even greater contamination. With the increased popularity of burning wood for household heating purposes, the environmental health hazard of burning CCA-treated wood needs recognition and evaluation. The role of chromium and copper in contributing to these health problems is conjectural. We would suggest that all three elements could be responsible for this kaleidoscopic clinical pattern.

Joy Savides Felker provided advice and secretarial aid and Lee Sjouik, MS, contributed technical assistance.

References

1. Oettingen WF: Poisoning—A Guide to Clinical Diagnosis and Treatment, ed 2. Philadelphia, WB Saunders Co, 1958, pp 239–242.

2. Woolson E, Ahronson N: Separation and detection of arsenicals pesticides residues and some of their metabolites by high pressure liquid chromatography-graphite furnace atomic absorption spectro. J Assoc Off Anal Chem 1980;63:523–528.

3. Boylen GW, Hardy NL: Distribution of arsenic in nonexposed persons (hair, liver, and urine). Am Ind Hyg Assoc J 1967;28:148–150.

4. Mees RA: The nails with arsenical polyneuritis. JAMA 1919;72:1337.

5. Biologic and economic assessment of pentachlorophenol inorganic arsenicals, and creosote, in Wood Preservatives, Technical Bulletin No. 1658–1, US Dept of Agriculture, 1980, vol 1, pp 31–192.

6. Tseng WP: Effects and dose-response relationships of skin cancer and blackfoot disease with arsenic. Environ Health Perspect 1977; 19:109–119.

7. Stewart CP, Stolman A: Toxicology—Mechanisms and Analytical Methods. New York, Academic Press Inc, 1960, vol 1, pp 202–206.

8. Moeschlin S: Poisoning—Diagnosis and Treatment New York, Grune & Stratton Inc, 1965, pp 162–169.

9. Creason JP, Hinners TA, Bumgarner SE, et al: Trace elements in hair as related to exposure in metropolitan New York. Clin Chem 1975; 21:603–612.

10. Harrison WW, Yurachek JP, Benson CA: The determination of trace elements in human hair by atomic absorption spectroscopy. Clin Chim Acta 1969;23:83–91.

11. Harrison WW, Clemena GG: Survey analysis of trace elements in human fingernails by spark source mass spectrometry. Clin Chim Acta 1971;36:485–492.

12. Harrison WW, Tyree AB: The determination of trace elements in human fingernails by atomic absorption spectroscopy. Clin Chim Acta 1971;31:63–73.

13. Casarett LJ, Doull J: The Basic Science of Poisons Toxicology. New York, Macmillan Publishing Co Inc, 1975, pp 464–465.

14. Ginsburg JM: Renal mechanism for excretion and transformation of arsenic in the dog. Am J Physiol 1965;208:832–840.

15. Feldman RG, Niles CA, Kelly-Hayes M, et al: Peripheral neuropathy in arsenic smelter workers. Neurology 1979;29:939–944.

16. International Agency for Research on Cancer (IARC): Arsenic and Arsenic Compounds, IARC monographs on the evaluation of the carcinogenic risk of chemicals to humans. Lyon, France, World Health Organization, IRAC, vol 23, pp 39–141.

17. Woolson EA: Fate of arsenicals in different environmental substrates. Environ Health Perspect 1977;19:73–81.

18. Fowler BA: International conference on environmental arsenic: An overview. Environ Health Perspect 1977;19:239–242.



The National Academies | 500 Fifth St. N.W. | Washington, D.C. 20001
Copyright © National Academy of Sciences. All rights reserved.
Terms of Use and Privacy Statement